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Acute infero-postero myocardial infarction.
<bold>Figure 24-7. Pathogenesis of acutecoronary syndromes. A.</bold> A normal coronary artery has an intact endothelium surrounded by smooth muscle cells. <bold>B.</bold> Endothelial cell activation or injury recruits monocytes and T lymphocytes to the site of injury, leading to development of a fatty streak. <bold>C.</bold> Continued oxidative stress within a fatty streak leads to development of an atherosclerotic plaque. <bold>D.</bold> Macrophage apoptosis and co...
<bold>Figure 24-7. Pathogenesis of acutecoronary syndromes. A.</bold> A normal coronary artery has an intact endothelium surro...
Acute anterior myocardial infarction. Transmural injury is manifested by ST-segment elevation of greater than 0.2 mV in leads V1 to V3.
Myocardial infarction, ST elevation.
<bold>FIGURE 26-10</bold> (<bold>Top</bold>) (<bold>A</bold>) ECG tracing showing normal P, Q,R,S, and T waves. (<bold>B</bold>) ST elevation with acute ischemia. (<bold>C</bold>) Q wave with acute myocardial infarction. (<bold>Bottom</bold>) Current-of-injury patterns with acute ischemia. With predominant subendocardial ischemia (<bold>A</bold>), the resultant ST segment is directed toward the inner layer of the affected ventricle and the ventricular cavity. Overlying leads therefore record ST-segment depression. With ischemia inv...
<bold>FIGURE 26-10</bold> (<bold>Top</bold>) (<bold>A</bold>) ECG tracing showing normal P, Q,R,S, and ...
Acute myocardial infarct. A cross-section of the ventricles of a man who died a few days after the onset of severe chest pain shows a transmural infarct in the posterior and septal regions of the left ventricle. The necrotic myocardium is soft, yellowish, and sharply demarcated.
Acute myocardial infarct. A cross-section of the ventricles of a man who died a few days after the onset of severe chest pain shows a trans...