Peritonitis, Acute

Diverticulosis. Diverticulosis seen in a segment of the descending colon.

Figure 41.3. CT appearance of appendicitis.

FIGURE 118.1. Perforated appendicitis with abscess and fecalith. The upright abdominal roentgenogram shows numerous dilated loops of bowel and a calcified fecalith <italic>(arrow).</bold> Note that the space between the individual loops indicates the presence of intraperitoneal fluid.

FIGURE 118.2. Perforated appendicitis with abscess and fecalith. Ultrasonography of the pelvis shows a complex mass <italic>(A)</bold> with a fecalith <italic>(arrow)</bold> producing characteristic acoustic shadowing to the right of the bladder <italic>(B)</bold>.

<bold>Figure 8.64. Appendicitis. A.</bold> CT image shows a fecalith in a thickened appendix (<i>arrow</i>). <bold>B.</bold> Image slightly higher shows the thickened appendiceal wall (<i>arrow</i>).

<bold><italic>Figure 15-14</bold> Perforated peptic ulcer of stomach.</bold>

<bold><italic>Figure 15-26</bold> Colonic diverticulosis and diverticulitis. A,</bold> Complications of diverticular inflammation (diverticulitis). <bold>B,</bold> Detail of the anatomy of an acquired diverticulum.

<bold><italic>Figure 15-33</bold> Acute appendicitis.</bold> A fecalith is wedged into the mouth of the appendix (left).

Gastric ulcer. The stomach has been opened to reveal a sharply demarcated, deep peptic ulcer on the lesser curvature.

Duodenal ulcer. A sharply punched-out peptic ulcer of the duodenum is situated immediately below the pylorus.

Chronic hepatitis with cirrhosis. A photomicrograph of the liver from a patient with long-standing chronic active hepatitis B shows hepatocellular nodules and chronically inflamed fibrous septa.

<bold>Fig GI 10-10 Peptic ulcer disease.</bold> (A) Large incisura (black arrow) simulating a filling defect on the greater curvature. The incisura is incited by a long ulcer (white arrows) on the lesser curvature. (B) Double pylorus. The true pylorus and the accessory channel along the lesser curvature are separated by a bridge, or septum, that produces the appearance of a discrete lucent filling defect (arrow).

<bold>Fig GI 10-10 Peptic ulcer disease.</bold> (A) Large incisura (black arrow) simulating a filling defect on the greater curvature. The incisura is incited by a long ulcer (white arrows) on the lesser curvature. (B) Double pylorus. The true pylorus and the accessory channel along the lesser curvature are separated by a bridge, or septum, that produces the appearance of a discrete lucent filling defect (arrow).

Figure 18-79<bold>BARIUM ENEMA, DIVERTICULAR DISEASE.</bold>Double-contrast barium enema demonstrates multiple outpouchings (diverticula) within the sigmoid colon (<italic>arrows</bold>).<bold><italic>COMMENT:</bold></bold>These diverticula are round and smooth, without evidence of barium extra vasation. Diverticulitis can be associated with the se complicating changes.

<bold>FIGURE 39-6</bold> (<bold>Top left</bold>) Location of diverticula in the sigmoid colon. (<bold>Top right</bold>) A portion of the sigmoid colon, showing the haustra and teniae coli. (<bold>Bottom left</bold>) Diverticulosis. (<bold>Bottom right</bold>) Diverticulitis.

<bold>FIGURE 117.11</bold> Ultrasonographic appearance of appendicitis. The appendix appears as a thick, noncompressible tubular structure with central hypoechogenicity. The walls are 5 mm thick, over the 3-mm limit for a normal appendix. (Courtesy of Dr. Beverly Coleman, Department of Radiology, Hospital of the University of Pennsylvania, Philadelphia, PA.)

Alcoholic cirrhosis. The surface of the liver displays innumerable small, regular nodules.

Alcoholic cirrhosis. A photomicrograph shows small regular nodules surrounded by uniform fibrous septa.

Cirrhosis of the liver. The consequence of chronic hepatic injury is the formation of regenerating nodules separated by bands of fibrous connective tissue (blue).